[1]
|
李晓景, 高孝忠, 褚衍六, 等. 结直肠癌合并高风险性腺瘤内镜治疗时机的临床研究[J]. 中华胃肠内镜电子杂志, 2021, 8(1): 13-17.
|
[2]
|
Nakamura, F., Sato, Y., Okamoto, K., et al. (2022) Colorectal Carcinoma Occurring via the Adenoma-Carcinoma Pathway in Patients with Serrated Polyposis Syndrome. Journal of Gastroenterology, 57, 286-299. https://doi.org/10.1007/s00535-022-01858-8
|
[3]
|
Baidoun, F., Elshiwy, K., Elkeraie, Y., et al. (2021) Colorectal Cancer Epidemiology: Recent Trends and Impact on Outcomes. Current Drug Targets, 22, 998-1009. https://doi.org/10.2174/18735592MTEx9NTk2y
|
[4]
|
吴艳惠, 杨鑫, 吴现瑞. 代谢综合征与结直肠癌相关性的研究进展[J]. 中国肿瘤外科杂志, 2023, 15(3): 262-265.
|
[5]
|
Breau, G. and Ellis, U. (2020) Risk Factors Associated with Young-Onset Colorectal Adenomas and Cancer: A Systematic Review and Meta-Analysis of Observational Research. Cancer Control, 27. https://doi.org/10.1177/1073274820976670
|
[6]
|
Fliss-Isakov, N., Zelber-Sagi, S., Webb, M., et al. (2017) Distinct Metabolic Profiles Are Associated with Colorectal Adenomas and Serrated Polyps. Obesity, 25, S72-S80. https://doi.org/10.1002/oby.22001
|
[7]
|
Hirode, G. and Wong, R, J. (2020) Trends in the Prevalence of Metabolic Syndrome in the United States, 2011-2016. JAMA, 323, 2526-2528. https://doi.org/10.1001/jama.2020.4501
|
[8]
|
钏莉雪, 常江, 赵锦涵. 代谢综合征与结直肠腺瘤性息肉的相关性研究[J]. 胃肠病学, 2019, 24(11): 699-702.
|
[9]
|
Ozkan, N.T., Tokmak, A., Guzel, A.I., et al. (2015) The Association between Endometrial Polyps and Metabolic Syndrome: A Case-Control Study. Australian and New Zealand Journal of Obstetrics and Gynaecology, 55, 274-278. https://doi.org/10.1111/ajo.12339
|
[10]
|
Bueloni-Dias, F.N., Spadoto-Dias, D., Delmanto, L.R., et al. (2016) Metabolic Syndrome as a Predictor of Endometrial Polyps in Postmenopausal Women. Menopause, 23, 759-764. https://doi.org/10.1097/GME.0000000000000616
|
[11]
|
Chen, Y., Wang, T., Gao, R., et al. (2023) Effects of Metabolic Syndrome and Its Components on the Postoperative Recurrence in Chronic Rhinosinusitis with Nasal Polyps’ Patients. Brazilian Journal of Otorhinolaryngology, 90, Article ID: 101371. https://doi.org/10.1016/j.bjorl.2023.101371
|
[12]
|
Jia, W.P., Xiang, K.S., Chen, L., et al. (2002) Epidemiological Study on Obesity and Its Comorbidities in Urban Chinese Older Than 20 Years of Age in Shanghai, China. Obesity Reviews, 3, 157-165. https://doi.org/10.1046/j.1467-789X.2002.00071.x
|
[13]
|
Moon, J.M., Im, J.P., Kim, D., et al. (2021) Increasing Changes in Visceral Adiposity Is Associated with Higher Risk for Colorectal Adenoma: Multilevel Analysis in a Prospective Cohort. Journal of Gastroenterology and Hepatology, 36, 1836-1842. https://doi.org/10.1111/jgh.15364
|
[14]
|
Jung, I.S., Shin, C.M., Park, S.J., et al. (2019) Association of Visceral Adiposity and Insulin Resistance with Colorectal Adenoma and Colorectal Cancer. Intestinal Research, 17, 404-412. https://doi.org/10.5217/ir.2018.00072
|
[15]
|
吴文琪, 万远太. 结直肠息肉发生发展相关因素的研究现状[J]. 消化肿瘤杂志(电子版), 2021, 13(2): 148-152.
|
[16]
|
Kasprzak, A. (2021) Insulin-Like Growth Factor 1 (IGF-1) Signaling in Glucose Metabolism in Colorectal Cancer. International Journal of Molecular Sciences, 22, Article 6434. https://doi.org/10.3390/ijms22126434
|
[17]
|
Alagaratnam, S., Loizidou, M., Yang, S.Y., et al. (2020) Increased Expression of IGF-1Ec with Increasing Colonic Polyp Dysplasia and Colorectal Cancer. Journal of Cancer Research and Clinical Oncology, 146, 2861-2870. https://doi.org/10.1007/s00432-020-03345-0
|
[18]
|
Qin, M., Wang, H.P., Song, B., et al. (2021) [Relationship between Insulin Resistance, Serum VCAM-1, FGF19, IGF-1 and Colorectal Polyps]. Chinese Journal of Oncology, 43, 553-562.
|
[19]
|
Brown, J.C. (1982) Gastric Inhibitory Polypeptide. Springer, Berlin. https://doi.org/10.1007/978-3-642-81771-7_1
|
[20]
|
Dupre, J., Ross, S, A., Watson, D., et al. (1973) Stimulation of Insulin Secretion by Gastric Inhibitory Polypeptide in Man. The Journal of Clinical Endocrinology & Metabolism, 37, 826-828. https://doi.org/10.1210/jcem-37-5-826
|
[21]
|
Sasaki, Y., Takeda, H., Sato, T., et al. (2011) Increased Levels of Serum Glucose-Dependent Insulinotropic Polypeptide as a Novel Risk Factor for Human Colorectal Adenoma. Metabolism, 60, 1253-1258. https://doi.org/10.1016/j.metabol.2011.01.007
|
[22]
|
Rogers, M., Gill, D., Ahlqvist, E., et al. (2023) Genetically Proxied Impaired GIPR Signaling and Risk of 6 Cancers. iScience, 26, Article ID: 106848. https://doi.org/10.1016/j.isci.2023.106848
|
[23]
|
Chen, F.P., Wang, H.M., Chiang, F.F., et al. (2014) The Metabolic Syndrome Is Associated with an Increased Risk of Colorectal Polyps Independent of Plasma Homocysteine. Annals of Nutrition and Metabolism, 64, 106-112. https://doi.org/10.1159/000363418
|
[24]
|
Zhang, R., Yin, J., Huo, C., et al. (2022) The Relationship between Colorectal Polyps and Serum Lipid Levels: A Systematic Review and Meta-Analysis. Journal of Clinical Gastroenterology, 56, 654-667. https://doi.org/10.1097/MCG.0000000000001678
|
[25]
|
Tang, C.T., Li, J., Yang, Z., et al. (2022) Comparison of Some Biochemical Markers between Early-Onset and Late-Onset Colorectal Precancerous Lesions: A Single-Center Retrospective Study. Journal of Clinical Laboratory Analysis, 36, e24637. https://doi.org/10.1002/jcla.24637
|
[26]
|
Crespo-Sanjuan, J., Calvo-Nieves, M.D., Aguirre-Gervas, B., et al. (2015) Early Detection of High Oxidative Activity in Patients with Adenomatous Intestinal Polyps and Colorectal Adenocarcinoma: Myeloperoxidase and Oxidized Low-Density Lipoprotein in Serum as New Markers of Oxidative Stress in Colorectal Cancer. Laboratory Medicine, 46, 123-135. https://doi.org/10.1309/LMZJJU6BC86WUDHW
|
[27]
|
Chen, H., Zhou, H., Liang, Y., et al. (2023) UHPLC-HRMS-Based Serum Untargeted Lipidomics: Phosphatidylcholines and Sphingomyelins Are the Main Disturbed Lipid Markers to Distinguish Colorectal Advanced Adenoma from Cancer. Journal of Pharmaceutical and Biomedical Analysis, 234, Article ID: 115582. https://doi.org/10.1016/j.jpba.2023.115582
|
[28]
|
Zhu, Y., Wang, L., Nong, Y., et al. (2021) Serum Untargeted UHPLC-HRMS-Based Lipidomics to Discover the Potential Biomarker of Colorectal Advanced Adenoma. Cancer Management and Research, 13, 8865-8878. https://doi.org/10.2147/CMAR.S336322
|
[29]
|
Chen, H.Y., Lee, W.H., Hsu, H.L., et al. (2022) Arterial Stiffness Is Associated with High-Risk Colorectal Adenomas and Serrated Lesions: A Cross-Sectional Study in a Taiwanese Population. Journal of Cardiology, 80, 139-144. https://doi.org/10.1016/j.jjcc.2022.03.013
|
[30]
|
Yamaji, Y., Mitsushima, T. and Koike, K. (2014) Pulse-Wave Velocity, the Ankle-Brachial Index, and the Visceral Fat Area Are Highly Associated with Colorectal Adenoma. Digestive and Liver Disease, 46, 943-949. https://doi.org/10.1016/j.dld.2014.05.012
|
[31]
|
Boden, G., Homko, C., Barrero, C.A., et al. (2015) Excessive Caloric Intake Acutely Causes Oxidative Stress, GLUT4 Carbonylation, and Insulin Resistance in Healthy Men. Science Translational Medicine, 7, 304re7. https://doi.org/10.1126/scitranslmed.aac4765
|
[32]
|
Xu, H., Li, X., Adams, H., et al. (2018) Etiology of Metabolic Syndrome and Dietary Intervention. International Journal of Molecular Sciences, 20, Article 128. https://doi.org/10.3390/ijms20010128
|
[33]
|
Boldogh, I., Hajas, G., Aguilera-Aguirre, L., et al. (2012) Activation of Ras Signaling Pathway by 8-Oxoguanine DNA Glycosylase Bound to Its Excision Product, 8-Oxoguanine. Journal of Biological Chemistry, 287, 20769-20773. https://doi.org/10.1074/jbc.C112.364620
|
[34]
|
German, P., Szaniszlo, P., Hajas, G., et al. (2013) Activation of Cellular Signaling by 8-Oxoguanine DNA Glycosylase-1-Initiated DNA Base Excision Repair. DNA Repair, 12, 856-863. https://doi.org/10.1016/j.dnarep.2013.06.006
|
[35]
|
Wang, T., Brown, N.M., McCoy, A.N., et al. (2022) ω-3 Polyunsaturated Fatty Acids, Gut Microbiota, Microbial Metabolites, and Risk of Colorectal Adenomas. Cancers, 14, Article 4443. https://doi.org/10.3390/cancers14184443
|
[36]
|
Finucane, O.M., Lyons, C.L., Murphy, A.M., et al. (2015) Monounsaturated Fatty Acid-Enriched High-Fat Diets Impede Adipose NLRP3 Inflammasome-Mediated IL-1β Secretion and Insulin Resistance Despite Obesity. Diabetes, 64, 2116-2128. https://doi.org/10.2337/db14-1098
|
[37]
|
Wang, J., Tang, H., Zhang, C., et al. (2015) Modulation of Gut Microbiota during Probiotic-Mediated Attenuation of Metabolic Syndrome in High Fat Diet-Fed Mice. The ISME Journal, 9, 1-15. https://doi.org/10.1038/ismej.2014.99
|
[38]
|
Hulston, C.J., Churnside, A.A. and Venables, M.C. (2015) Probiotic Supplementation Prevents High-Fat, Overfeeding-Induced Insulin Resistance in Human Subjects. British Journal of Nutrition, 113, 596-602. https://doi.org/10.1017/S0007114514004097
|
[39]
|
Chassaing, B., Miles-Brown, J., Pellizzon, M., et al. (2015) Lack of Soluble Fiber Drives Diet-Induced Adiposity in Mice. American Journal of Physiology-Gastrointestinal and Liver Physiology, 309, G528-G541. https://doi.org/10.1152/ajpgi.00172.2015
|
[40]
|
Yan, H., Potu, R., Lu, H., et al. (2013) Dietary Fat Content and Fiber Type Modulate Hind Gut Microbial Community and Metabolic Markers in the Pig. PLOS ONE, 8, e59581. https://doi.org/10.1371/journal.pone.0059581
|
[41]
|
Gutting, T., Weber, C.A., Weidner, P., et al. (2018) PPARgamma-Activation Increases Intestinal M1 Macrophages and Mitigates Formation of Serrated Adenomas in Mutant KRAS Mice. Oncoimmunology, 7, e1423168. https://doi.org/10.1080/2162402X.2017.1423168
|
[42]
|
Kurnaz-Gomleksiz, O., Torun, B.C., Isbir, T., et al. (2022) The Role of PPAR-Gamma C161T Polymorphism in Colorectal Cancer Susceptibility. In Vivo, 36, 1911-1915. https://doi.org/10.21873/invivo.12911
|
[43]
|
Barry, E.L., Fedirko, V., Uppal, K., et al. (2020) Metabolomics Analysis of Aspirin’s Effects in Human Colon Tissue and Associations with Adenoma Risk. Cancer Prevention Research, 13, 863-876. https://doi.org/10.1158/1940-6207.CAPR-20-0014
|
[44]
|
Phua, L.C., Chue, X.P., Koh, P.K., et al. (2014) Non-Invasive Fecal Metabonomic Detection of Colorectal Cancer. Cancer Biology & Therapy, 15, 389-397. https://doi.org/10.4161/cbt.27625
|
[45]
|
Denkert, C., Budczies, J., Weichert, W., et al. (2008) Metabolite Profiling of Human Colon Carcinoma—Deregulation of TCA Cycle and Amino Acid Turnover. Molecular Cancer, 7, Article No. 72. https://doi.org/10.1186/1476-4598-7-72
|
[46]
|
Hirayama, A., Kami, K., Sugimoto, M., et al. (2009) Quantitative Metabolome Profiling of Colon and Stomach Cancer Microenvironment by Capillary Electrophoresis Time-of-Flight Mass Spectrometry. Cancer Research, 69, 4918-4925. https://doi.org/10.1158/0008-5472.CAN-08-4806
|
[47]
|
Weir, T.L., Manter, D.K., Sheflin, A.M., et al. (2013) Stool Microbiome and Metabolome Differences between Colorectal Cancer Patients and Healthy Adults. PLOS ONE, 8, e70803. https://doi.org/10.1371/journal.pone.0070803
|
[48]
|
Wang, X., Wang, J., Rao, B. and Deng, L. (2022) Gut Flora Profiling and Fecal Metabolite Composition of Colorectal Cancer Patients and Healthy Individuals. Experimental and Therapeutic Medicine, 23, Article No. 250. https://doi.org/10.3892/etm.2022.11175
|
[49]
|
Yang, M., Liu, S. and Zhang, C. (2022) The Related Metabolic Diseases and Treatments of Obesity. Healthcare, 10, Article 1616. https://doi.org/10.3390/healthcare10091616
|