[1]
|
Roth, G.A., Mensah, G.A., Johnson, C.O., et al. (2020) Global Burden of Cardiovascular Diseases and Risk Factors, 1990-2019: Update from the GBD 2019 Study. Journal of the American College of Cardiology, 76, 2982-3021.
https://doi.org/10.1016/j.jacc.2020.11.010
|
[2]
|
Jiang, F., Chen, Q., Wang, W., et al. (2020) Hepatocyte Derived Extracellular Vesicles Promote Endothelial Inflammation and Atherogenesis via microRNA-1. Journal of Hepatology, 72, 156-166.
https://doi.org/10.1016/j.jhep.2019.09.014
|
[3]
|
Nasiri-Ansari, N., Androutsakos, T., Flessa, C.M., et al. (2022) Endothelial Cell Dysfunction and Nonalcoholic Fatty Liver Disease (NAFLD): A Concise Review. Cells, 11, Article No. 2511. https://doi.org/10.3390/cells11162511
|
[4]
|
Osonoi, Y., Mita, T., Azuma, K., Nakajima, K., Masuyama, A., Goto, H., et al. (2018) Defective Autophagy in Vascular Smooth Muscle Cells Enhances Cell Death and Atherosclerosis. Autophagy, 14, 1991-2006.
https://doi.org/10.1080/15548627.2018.1501132
|
[5]
|
Libby, P., Ridker, P.M. and Hansson, G.K. (2011) Progress and Challenges in Translating the Biology of Atherosclerosis. Nature, 473, 317-325. https://doi.org/10.1038/nature10146
|
[6]
|
Kadlec, A.O., Chabowski, D.S., Ait-Aissa, K., et al. (2016) Role of PGC-1α in Vascular Regulation: Implications for Atherosclerosis. Arteriosclerosis, Thrombosis, and Vascular Biology, 36, 1467-1474.
https://doi.org/10.1161/ATVBAHA.116.307123
|
[7]
|
Gimbrone, M.A. and García-Cardeña, G. (2016) Endothelial Cell Dysfunction and the Pathobiology of Atherosclerosis. Circulation Research, 118, 620-636. https://doi.org/10.1161/CIRCRESAHA.115.306301
|
[8]
|
Heitzer, T., Schlinzig, T., Krohn, K., et al. (2001) Endo-thelial Dysfunction, Oxidative Stress, and Risk of Cardiovascular Events in Patients with Coronary Artery Disease. Cir-culation, 104, 2673-2678.
https://doi.org/10.1161/hc4601.099485
|
[9]
|
刘梨, 龚后武, 张建影, 等. 中医药治疗颈动脉粥样硬化斑块临床疗效的Meta分析[J]. 中医药学报, 2015, 43(3): 20-24.
|
[10]
|
赵立凤, 于红红, 田维毅. 中药单体调控血管内皮细胞自噬干预动脉粥样硬化的研究进展[J]. 中华中医药学刊, 2021, 39(11): 117-120.
|
[11]
|
柴钰, 金翠柳, 凌望, 等. 血管内皮细胞焦亡在动脉粥样硬化发生、发展中的作用及机制研究进展[J]. 医学研究杂志, 2021, 50(9): 147-150+172.
|
[12]
|
雷紫琴, 栾飞, 高铭, 等. 细胞焦亡与心血管疾病的关系及中医药防治研究进展[J]. 中国中药杂志, 2023, 48(7): 1779-1791.
|
[13]
|
Kong, P., Cui, Z.-Y., Huang, X.-F., et al. (2022) Inflammation and Atheroscle-rosis: Signaling Pathways and Therapeutic Intervention. Signal Transduction and Targeted Therapy, 7, Article No. 131.
https://doi.org/10.1038/s41392-022-00955-7
|
[14]
|
Ley, K., Miller, Y.I. and Hedrick, C.C. (2011) Monocyte and Macrophage Dynamics during Atherogenesis. Arteriosclerosis, Thrombosis, and Vascular Biology, 31, 1506-1516. https://doi.org/10.1161/ATVBAHA.110.221127
|
[15]
|
李容, 王新文, 杨晓辉, 等. 细胞命运之终点——细胞死亡[J]. 科学通报, 2016, 61(18): 1983-1987.
|
[16]
|
Grootjans, S., Vanden, Berghe, T. and Vandenabeele, P. (2017) Initia-tion and Execution Mechanisms of Necroptosis: An Overview. Cell Death & Differentiation, 24, 1184-1195. https://doi.org/10.1038/cdd.2017.65
|
[17]
|
Xing, S.-S., Yang, J., Li, W., et al. (2020) Salidroside Decreases Athero-sclerosis Plaque Formation via Inhibiting Endothelial Cell Pyroptosis. Inflammation, 43, 433-440. https://doi.org/10.1007/s10753-019-01106-x
|
[18]
|
Zhang, Y., Chen, X., Gueydan, C. and Han, J. (2018) Plasma Membrane Changes during Programmed Cell Deaths. Cell Research, 28, 9-21. https://doi.org/10.1038/cr.2017.133
|
[19]
|
赵为陈, 江俊麟. 细胞焦亡在血管内皮细胞功能障碍中的研究进展[J]. 重庆医学, 2020, 49(15): 2577-2582.
|
[20]
|
Liu, X., Zhang, Z., Ruan, J., et al. (2016) Inflammasome-Activated Gasdermin D Causes Pyroptosis by Forming Membrane Pores. Nature, 535, 153-158. https://doi.org/10.1038/nature18629
|
[21]
|
杜会君, 杨红晓, 白俊彩, 等. 细胞焦亡与心血管疾病[J]. 国际心血管病杂志, 2022, 49(2): 92-95.
|
[22]
|
刘孜琦, 聂妍琦, 傅旖灵, 等. 基于NLRP3炎症小体探讨电针对脑缺血再灌注损伤大鼠细胞焦亡的影响[J]. 中国中医药信息杂志, 2022, 29(10): 84-89.
|
[23]
|
Guo, H., Callaway, J.B. and Ting, J.P.Y. (2015) Inflammasomes: Mechanism of Action, Role in Disease, and Therapeutics. Nature Medicine, 21, 677-687. https://doi.org/10.1038/nm.3893
|
[24]
|
Chen, Y., He, H., Jiang, H., et al. (2020) Discovery and Optimization of 4-Oxo-2-thioxo-thiazolidinones as NOD-Like Receptor (NLR) Family, Pyrin Domain-Containing Protein 3 (NLRP3) Inhibitors. Bioorganic & Medicinal Chemistry Letters, 30, Article ID: 127021. https://doi.org/10.1016/j.bmcl.2020.127021
|
[25]
|
Yin, Y., Li, X., Sha, X., et al. (2015) Early Hyperlipidemia Pro-motes Endothelial Activation via a Caspase-1-Sirtuin 1 Pathway. Arteriosclerosis, Thrombosis, and Vascular Biology, 35, 804-816.
https://doi.org/10.1161/ATVBAHA.115.305282
|
[26]
|
赵权威, 李辉, 刘大男, 等. 达格列净减轻氧化低密度脂蛋白诱导的内皮细胞焦亡和功能障碍[J]. 中国组织工程研究, 2024, 28(1): 80-85.
|
[27]
|
Heeschen, C., Jang, J.J., Weis, M., et al. (2001) Nicotine Stimulates Angiogenesis and Promotes Tumor Growth and Atherosclerosis. Nature Medicine, 7, 833-839. https://doi.org/10.1038/89961
|
[28]
|
Wu, X., Zhang, H., Qi, W., et al. (2018) Nicotine Pro-motes Atherosclerosis via ROS-NLRP3-Mediated Endothelial Cell Pyroptosis. Cell Death & Disease, 9, Article No. 171. https://doi.org/10.1038/s41419-017-0257-3
|
[29]
|
Jiang, X., Ma, C., Gao, Y., et al. (2023) Tongxinluo Attenuates Atherosclerosis by Inhibiting ROS/NLRP3/Caspase-1- Mediated Endothelial Cell Pyroptosis. Journal of Ethnopharma-cology, 304, Article ID: 116011.
https://doi.org/10.1016/j.jep.2022.116011
|
[30]
|
袁育林. 姜黄素抑制NLRP3介导的细胞焦亡改善内皮细胞功能[D]: [硕士学位论文]. 衡阳: 南华大学, 2022.
|
[31]
|
杨佳乐, 沈祥春. 灯盏花乙素通过抑制NLRP3/caspase-1信号通路改善LPS+ATP诱导内皮细胞炎症反应和细胞焦亡[J]. 中国药理学通报, 2022, 38(8): 1196-1201.
|
[32]
|
吴树宁, 王凯, 施思, 等. 大黄素预处理对LPS/ATP诱导的人脐静脉内皮细胞焦亡的影响[J]. 山西医科大学学报, 2019, 50(4): 410-414.
|
[33]
|
张艳, 周庆兵, 徐凤芹. 栀子苷防治动脉粥样硬化的分子生物学分析[J]. 世界中西医结合杂志, 2021, 16(8): 1373-1378.
|
[34]
|
王复婧, 黄小飞, 赵丹丹, 等. 栀子苷通过PI3K通路抑制OX-LDL诱导HUVEC中KLF2表达[J]. 中药药理与临床, 2018, 34(1): 53-56.
|
[35]
|
He, J., Deng, Y., Ren, L., et al. (2023) Isoliquiritigenin from Licorice Flavonoids Attenuates NLRP3-Mediated Pyroptosis by SIRT6 in Vascular Endothelial Cells. Journal of Ethnopharmacology, 303, Article ID: 115952.
https://doi.org/10.1016/j.jep.2022.115952
|