[1]
|
Cho, N.H., Shaw, J., Karuranga, S., et al. (2018) IDF Diabetes Atlas: Global Estimates of Diabetes Prevalence for 2017 and Projections for 2045. Diabetes Research and Clinical Practice, 138, 271-281.
https://doi.org/10.1016/j.diabres.2018.02.023
|
[2]
|
Bruno, G., Runzo, C., Cavallo-Perin, P., et al. (2005) Incidence of Type 1 and Type 2 Diabetes in Adults Aged 30-49 Years: The Population-Based Registry in the Province of Turin, It-aly. Diabetes Care, 28, 2613-2619.
https://doi.org/10.2337/diacare.28.11.2613
|
[3]
|
Holman, N., Young, B. and Gadsby, R. (2015) Current Prevalence of Type 1 and Type 2 Diabetes in Adults and Children in the UK. Diabetic Medicine, 9, 1119-1120. https://doi.org/10.1111/dme.12791
|
[4]
|
Group UPDS (1998) Tight Blood Pressure Control and Risk of Macro-vascular and Microvascular Complications in Type 2 Diabetes: UKPDS 38. BMJ, 317, 703-713. https://doi.org/10.1136/bmj.317.7160.703
|
[5]
|
Schmidt, A.M. (2019) Diabetes Mellitus and Cardiovascular Dis-ease: Emerging Therapeutic Approaches. Arteriosclerosis, Thrombosis, and Vascular Biology, 39, 558-568. https://doi.org/10.1161/ATVBAHA.119.310961
|
[6]
|
Zelnick, L.R., Weiss, N.S., Kestenbaum, B.R., et al. (2017) Diabetes and CKD in the United States Population, 2009- 2014. Clinical Journal of the American Society of Nephrology, 12, 1984-1990. https://doi.org/10.2215/CJN.03700417
|
[7]
|
Tuttle, K.R., Bakris, G.L., Bilous, R.W., et al. (2014) Diabetic Kidney Disease: A Report from an ADA Consensus Conference. Diabetes Care, 37, 2864-2883. https://doi.org/10.2337/dc14-1296
|
[8]
|
Yau, J.W., Rogers, S.L., Kawasaki, R., et al. (2012) Global Prevalence and Major Risk Factors of Diabetic Retinopathy. Diabetes Care, 35, 556-564. https://doi.org/10.2337/dc11-1909
|
[9]
|
Sabanayagam, C., Banu, R., Chee, M.L., et al. (2019) Incidence and Pro-gression of Diabetic Retinopathy: A Systematic Review. The Lancet Diabetes & Endocrinology, 7, 140-149. https://doi.org/10.1016/S2213-8587(18)30128-1
|
[10]
|
Boulton, A.J., Malik, R.A., Arezzo, J.C., et al. (2004) Dia-betic Somatic Neuropathies. Diabetes Care, 27, 1458-1486.
https://doi.org/10.2337/diacare.27.6.1458
|
[11]
|
Ang, L., Jaiswal, M., Martin, C., et al. (2014) Glucose Control and Diabetic Neuropathy: Lessons from Recent Large Clinical Trials. Current Diabetes Reports, 14, 1-15. https://doi.org/10.1007/s11892-014-0528-7
|
[12]
|
Albers, J.W. and Pop-Busui, R. (2014) Diabetic Neuropathy: Mechanisms, Emerging Treatments, and Subtypes. Current Neurology and Neuroscience Reports, 14, 1-11. https://doi.org/10.1007/s11910-014-0473-5
|
[13]
|
Volmer-Thole, M. and Lobmann, R. (2016) Neuropathy and Dia-betic Foot Syndrome. International Journal of Molecular Sciences, 17, 917. https://doi.org/10.3390/ijms17060917
|
[14]
|
Van Netten, J., Price, P.E., Lavery, L., et al. (2016) Prevention of Foot Ulcers in the At-Risk Patient with Diabetes: A Systematic Review. Diabetes/Metabolism Research and Reviews, 32, 84-98. https://doi.org/10.1002/dmrr.2701
|
[15]
|
Martin, E.T., Kaye, K.S., Knott, C., et al. (2016) Diabetes and Risk of Surgical Site Infection: A Systematic Review and Meta-Analysis. Infection Control & Hospital Epidemiology, 37, 88-99. https://doi.org/10.1017/ice.2015.249
|
[16]
|
Luk, A.O., Wu, H., Lau, E.S., et al. (2021) Temporal Trends in Rates of Infection-Related Hospitalisations in Hong Kong People with and without Diabetes, 2001-2016: A Retrospective Study. Diabetologia, 64, 109-118.
https://doi.org/10.1007/s00125-020-05286-2
|
[17]
|
Fang, M., Ishigami, J., Echouffo-Tcheugui, J.B., et al. (2021) Diabetes and the Risk of Hospitalisation for Infection: The Atherosclerosis Risk in Communities (ARIC) Study. Dia-betologia, 64, 2458-2465.
https://doi.org/10.1007/s00125-021-05522-3
|
[18]
|
Karter, A.J., Schillinger, D., Adams, A.S., et al. (2013) Elevated Rates of Diabetes in Pacific Islanders and Asian Subgroups: The Diabetes Study of Northern California (DISTANCE). Diabetes Care, 36, 574-579.
https://doi.org/10.2337/dc12-0722
|
[19]
|
Sun, H., Saeedi, P., Karuranga, S., et al. (2022) IDF Diabetes Atlas: Global, Regional and Country-Level Diabetes Prevalence Estimates for 2021 and Projections for 2045. Diabetes Research and Clinical Practice, 183, Article ID: 109119. https://doi.org/10.1016/j.diabres.2021.109119
|
[20]
|
Chen, D., Shen, J., Zhao, W., et al. (2017) Osteoarthritis: Toward a Comprehensive Understanding of Pathological Mechanism. Bone Re-search, 5, 1-13. https://doi.org/10.1038/boneres.2016.44
|
[21]
|
O’neill, T.W., Mccabe, P.S. and Mcbeth, J. (2018) Update on the Epidemiology, Risk Factors and Disease Outcomes of Osteoarthritis. Best Practice & Research Clinical Rheumatology, 32, 312-326.
https://doi.org/10.1016/j.berh.2018.10.007
|
[22]
|
黎丹东, 李琳琳, 苏峰, 等. 膝骨关节炎与性别和年龄的相关性研究[J]. 临床医学研究与实践, 2019, 4(31): 1-3+8.
https://doi.org/10.19347/j.cnki.2096-1413.201931001
|
[23]
|
Vina, E.R. and Kwoh, C.K. (2018) Epidemiology of Osteoarthritis: Literature Update. Current Opinion in Rheumatology, 30, 160. https://doi.org/10.1097/BOR.0000000000000479
|
[24]
|
Hunterdj, B. and Zeinstra, S. (1759) Osteoarthritis. The Lancet, 393, 1745.
https://doi.org/10.1016/S0140-6736(19)30417-9
|
[25]
|
Zhang, Z., Huang, C., Jiang, Q., et al. (2020) Guidelines for the Diagnosis and Treatment of Osteoarthritis in China (2019 Edition). Annals of Translational Medicine, 8, 1213.
|
[26]
|
Schett, G., Kleyer, A., Perricone, C., et al. (2013) Diabetes Is an Independent Predictor for Severe Osteoar-thritis: Results from a Longitudinal Cohort Study. Diabetes Care, 36, 403-409. https://doi.org/10.2337/dc12-0924
|
[27]
|
Eymard, F., Parsons, C., Edwards, M., et al. (2015) Diabetes Is a Risk Factor for Knee Osteoarthritis Progression. Osteoarthritis and Cartilage, 23, 851-859. https://doi.org/10.1016/j.joca.2015.01.013
|
[28]
|
Louati, K., Vidal, C., Berenbaum, F., et al. (2015) Association be-tween Diabetes Mellitus and Osteoarthritis: Systematic Literature Review and Meta-Analysis. RMD Open, 1, e000077. https://doi.org/10.1136/rmdopen-2015-000077
|
[29]
|
Abramoff, B. and Caldera, F.E. (2020) Osteoarthritis: Pathol-ogy, Diagnosis, and Treatment Options. Medical Clinics, 104, 293-311. https://doi.org/10.1016/j.mcna.2019.10.007
|
[30]
|
Li, G., Yin, J., Gao, J., et al. (2013) Subchondral Bone in Osteo-arthritis: Insight into Risk Factors and Microstructural Changes. Arthritis Research & Therapy, 15, 1-12. https://doi.org/10.1186/ar4405
|
[31]
|
Henrotin, Y., Pesesse, L. and Sanchez, C. (2012) Subchondral Bone and Oste-oarthritis: Biological and Cellular Aspects. Osteoporosis International, 23, 847-851. https://doi.org/10.1007/s00198-012-2162-z
|
[32]
|
Wenham, C.Y. and Conaghan, P.G. (2010) The Role of Synovitis in Osteoarthritis. Therapeutic Advances in Musculoskeletal Disease, 2, 349-359. https://doi.org/10.1177/1759720X10378373
|
[33]
|
Sharma, L., Pai, Y.C., Holtkamp, K., et al. (1997) Is Knee Joint Proprioception Worse in the Arthritic Knee versus the Unaffected Knee in Unilateral Knee Osteoarthritis? Arthritis & Rheumatism: Official Journal of the American College of Rheumatology, 40, 1518-1525. https://doi.org/10.1002/art.1780400821
|
[34]
|
Veronese, N., Cooper, C., Reginster, J.-Y., et al. (2019) Type 2 Dia-betes Mellitus and Osteoarthritis. Proceedings of the Seminars in Arthritis and Rheumatism, 49, 9-19. https://doi.org/10.1016/j.semarthrit.2019.01.005
|
[35]
|
王凯声, 亓建洪. 2型糖尿病与软骨退变相关性的研究进展[J]. 实用骨科杂志, 2021, 27(11): 1015-1018.
|
[36]
|
Buckwalter, J.A., Mankin, H.J. and Grodzinsky, A.J. (2005) Ar-ticular Cartilage and Osteoarthritis. American Academy of Orthopaedic Surgeons Instructional Course Lectures, 54, 465-480.
|
[37]
|
Glyn-Jones, S. and Palmer, A. (2015) Osteoarthritis. The Lancet, 386, 376-387.
https://doi.org/10.1016/S0140-6736(14)60802-3
|
[38]
|
Li, Z., Huang, Z. and Bai, L. (2021) Cell Interplay in Osteo-arthritis. Frontiers in Cell and Developmental Biology, 9, Article ID: 720477. https://doi.org/10.3389/fcell.2021.720477
|
[39]
|
Neumann, J., Hofmann, F.C., Heilmeier, U., et al. (2018) Type 2 Diabetes Patients Have Accelerated Cartilage Matrix Degeneration Compared to Diabetes Free Controls: Data from the Osteoarthritis Initiative. Osteoarthritis and Cartilage, 26, 751-761. https://doi.org/10.1016/j.joca.2018.03.010
|
[40]
|
Atayde, S.A., Yoshinari, N.H., Nascimento, D.P., et al. (2012) Experimental Diabetes Modulates Collagen Remodelling of Joints in Rats. Histology and Histopathology, 27, 1471-1479.
|
[41]
|
El Karib, A.O., Al-Ani, B., Al-Hashem, F., et al. (2016) Insulin and Vanadium Protect against Osteo-arthritis Development Secondary to Diabetes Mellitus in Rats. Archives of Physiology and Biochemistry, 122, 148-154.
https://doi.org/10.3109/13813455.2016.1159698
|
[42]
|
Rosa, S.C., Gonçalves, J., Judas, F., et al. (2009) Impaired Glucose Transporter-1 Degradation and Increased Glucose Transport and Oxidative Stress in Response to High Glucose in Chondrocytes from Osteoarthritic versus Normal Human Cartilage. Arthritis Research & Therapy, 11, R80. https://doi.org/10.1186/ar2713
|
[43]
|
Ribeiro, M., De Figueroa, P.L., Blanco, F., et al. (2016) Insulin Decreases Autophagy and Leads to Cartilage Degradation. Osteoarthritis and Cartilage, 24, 731-739. https://doi.org/10.1016/j.joca.2015.10.017
|
[44]
|
Vaamonde-Garcia, C., Courties, A., Pigenet, A., et al. (2017) The Nuclear Factor-Erythroid 2-Related Factor/Heme Oxygenase-1 Axis Is Critical for the Inflammatory Features of Type 2 Diabetes-Associated Osteoarthritis. Journal of Biological Chemistry, 292, 14505-14515. https://doi.org/10.1074/jbc.M117.802157
|
[45]
|
Nielen, J.T., De Vries, F., Dagnelie, P.C., et al. (2016) Use of Thi-azolidinediones and the Risk of Elective Hip or Knee Replacement: A Population Based Case-Control Study. British Journal of Clinical Pharmacology, 81, 370-378.
https://doi.org/10.1111/bcp.12786
|
[46]
|
马丁, 师东良, 李姣, 等. 关节软骨损伤再生修复研究进展[J]. 生命科学, 2021, 33(11): 1353-1362.
|
[47]
|
Rasheed, Z., Akhtar, N. and Haqqi, T.M. (2011) Advanced Glycation End Products Induce the Expression of Interleukin-6 and Interleukin-8 by Receptor for Advanced Glycation End Product-Mediated Ac-tivation of Mitogen-Activated Protein Kinases and Nuclear Factor-κB in Human Osteoarthritis Chondrocytes. Rheuma-tology, 50, 838-851.
https://doi.org/10.1093/rheumatology/keq380
|
[48]
|
Rasheed, Z. and Haqqi, T.M. (2012) Endoplasmic Reticulum Stress Induces the Expression of COX-2 through Activation of eIF2α, p38-MAPK and NF-κB in Advanced Glycation End Products Stimulated Human Chondrocytes. Biochimica et Biophysica Acta (BBA)-Molecular Cell Research, 1823, 2179-2189.
https://doi.org/10.1016/j.bbamcr.2012.08.021
|
[49]
|
Chen, Y.J., Sheu, M.L., Tsai, K.S., et al. (2013) Advanced Glycation End Products Induce Peroxisome Proliferator-Activated Receptor γ Down-Regulation-Related Inflammatory Signals in Human Chondrocytes via Toll-Like Receptor-4 and Receptor for Advanced Glycation End Products. PLOS ONE, 8, e66611.
https://doi.org/10.1371/journal.pone.0066611
|
[50]
|
Wang, J., Wang, G. and Sun, G. (2016) Role of PPARα in Down-Regulating AGE-Induced TGF-β and MMP-9 Expressions in Chondrocytes. Genetics and Molecular Research, 15, gmr7963. https://doi.org/10.4238/gmr.15027963
|
[51]
|
Nah, S.-S., Choi, I.-Y., Yoo, B., et al. (2007) Advanced Glycation End Products Increases Matrix Metalloproteinase-1, -3, and -13, and TNF-α in Human Osteoarthritic Chon-drocytes. FEBS Letters, 581, 1928-1932.
https://doi.org/10.1016/j.febslet.2007.03.090
|
[52]
|
Huang, C.-Y., Lai, K.-Y., Hung, L.-F., et al. (2011) Advanced Glycation End Products Cause Collagen II Reduction by Activating Janus Kinase/Signal Transducer and Activator of Transcription 3 Pathway in Porcine Chondrocytes. Rheumatology, 50, 1379-1389. https://doi.org/10.1093/rheumatology/ker134
|
[53]
|
Cecil, D.L., Johnson, K., Rediske, J., et al. (2005) Inflamma-tion-Induced Chondrocyte Hypertrophy Is Driven by Receptor for Advanced Glycation End Products. The Journal of Immunology, 175, 8296-8302.
https://doi.org/10.4049/jimmunol.175.12.8296
|
[54]
|
Trellu, S., Courties, A., Jaisson, S., et al. (2019) Impairment of Glyoxalase-1, an Advanced Glycation End-Product Detoxifying Enzyme, Induced by Inflammation in Age-Related Os-teoarthritis. Arthritis Research & Therapy, 21, 1-12.
https://doi.org/10.1186/s13075-018-1801-y
|
[55]
|
Chen, Y.J., Chan, D.C., Lan, K.C., et al. (2015) PPARγ Is In-volved in the Hyperglycemia-Induced Inflammatory Responses and Collagen Degradation in Human Chondrocytes and Diabetic Mouse Cartilages. Journal of Orthopaedic Research, 33, 373-381. https://doi.org/10.1002/jor.22770
|
[56]
|
中国2型糖尿病防治指南(2020年版)(下) [J]. 中国实用内科杂志, 2021, 41(9): 757-784.
|
[57]
|
中国2型糖尿病防治指南(2020年版)(上) [J]. 中国实用内科杂志, 2021, 41(8): 668-695.
|
[58]
|
Tchetina, E.V., Markova, G.A. and Sharapova, E.P. (2020) Insulin Resistance in Osteoarthritis: Similar Mechanisms to Type 2 Diabetes Mellitus. Journal of Nutrition and Metabolism, 2020, Article ID: 4143802.
https://doi.org/10.1155/2020/4143802
|
[59]
|
焦聚阳, 石晶晟, 夏军, 等. 糖尿病性骨关节炎发病机制研究进展[J]. 国际骨科学杂志, 2016, 37(6): 368-372.
|
[60]
|
Courties, A. and Sellam, J. (2016) Osteoarthritis and Type 2 Diabe-tes Mellitus: What Are the Links? Diabetes Research and Clinical Practice, 122, 198-206. https://doi.org/10.1016/j.diabres.2016.10.021
|
[61]
|
Hamada, D., Maynard, R., Schott, E., et al. (2016) Suppressive Effects of Insulin on Tumor Necrosis Factor-Dependent Early Osteoarthritic Changes Associated with Obesity and Type 2 Diabetes Mellitus. Arthritis & Rheumatology, 68, 1392-1402. https://doi.org/10.1002/art.39561
|
[62]
|
Sharma, A.R., Jagga, S., Lee, S.-S., et al. (2013) Interplay between Cartilage and Subchondral Bone Contributing to Pathogenesis of Osteoarthritis. International Journal of Molecular Sciences, 14, 19805-19830.
https://doi.org/10.3390/ijms141019805
|
[63]
|
Suri, S., Walsh, D.A. (2012) Osteochondral Alterations in Osteoar-thritis. Bone, 51, 204-211.
https://doi.org/10.1016/j.bone.2011.10.010
|
[64]
|
Siebelt, M., Waarsing, J., Groen, H., et al. (2014) Inhibited Osteo-clastic Bone Resorption through Alendronate Treatment in Rats Reduces Severe Osteoarthritis Progression. Bone, 66, 163-170.
https://doi.org/10.1016/j.bone.2014.06.009
|
[65]
|
Kazakia, G.J., Kuo, D., Schooler, J., et al. (2013) Bone and Carti-lage Demonstrate Changes Localized to Bone Marrow Edema-Like Lesions within Osteoarthritic Knees. Osteoarthritis and Cartilage, 21, 94-101.
https://doi.org/10.1016/j.joca.2012.09.008
|
[66]
|
Zhen, G., Wen, C., Jia, X., et al. (2013) Inhibition of TGF-β Sig-naling in Mesenchymal Stem Cells of Subchondral Bone Attenuates Osteoarthritis. Nature Medicine, 19, 704-712. https://doi.org/10.1038/nm.3143
|
[67]
|
Chen, Y., Wang, T., Guan, M., et al. (2015) Bone Turnover and Articular Cartilage Differences Localized to Subchondral Cysts in Knees with Advanced Osteoarthritis. Osteoarthritis and Carti-lage, 23, 2174-2183.
https://doi.org/10.1016/j.joca.2015.07.012
|
[68]
|
Patsch, J.M., Burghardt, A.J., Yap, S.P., et al. (2013) Increased Cortical Porosity in Type 2 Diabetic Postmenopausal Women with Fragility Fractures. Journal of Bone and Mineral Re-search, 28, 313-324.
https://doi.org/10.1002/jbmr.1763
|
[69]
|
Farr, J.N., Drake, M.T., Amin, S., et al. (2014) In Vivo Assessment of Bone Quality in Postmenopausal Women with Type 2 Diabetes. Journal of Bone and Mineral Research, 29, 787-795. https://doi.org/10.1002/jbmr.2106
|
[70]
|
Napoli, N., Strotmeyer, E.S., Ensrud, K.E., et al. (2014) Fracture Risk in Diabetic Elderly Men: The MrOS Study. Diabetologia, 57, 2057-2065. https://doi.org/10.1007/s00125-014-3289-6
|
[71]
|
Schwartz, A.V. (2016) Epidemiology of Fractures in Type 2 Dia-betes. Bone, 82, 2-8.
https://doi.org/10.1016/j.bone.2015.05.032
|
[72]
|
Jiajue, R., Jiang, Y., Wang, O., et al. (2014) Suppressed Bone Turnover Was Associated with Increased Osteoporotic Fracture Risks in Non-Obese Postmenopausal Chinese Women with Type 2 Diabetes Mellitus. Osteoporosis International, 25, 1999-2005. https://doi.org/10.1007/s00198-014-2714-5
|
[73]
|
Li, J., He, W., Liao, B., et al. (2015) FFA-ROS-P53-Mediated Mitochondrial Apoptosis Contributes to Reduction of Osteoblastogenesis and Bone Mass in Type 2 Diabetes Mellitus. Scientific Reports, 5, Article No. 12724.
https://doi.org/10.1038/srep12724
|
[74]
|
Reyes, C., Leyland, K.M., Peat, G., et al. (2016) Association between Overweight and Obesity and Risk of Clinically Diagnosed Knee, Hip, and Hand Osteoarthritis: A Population-Based Co-hort Study. Arthritis & Rheumatology, 68, 1869-1875. https://doi.org/10.1002/art.39707
|
[75]
|
Chen, Y., Huang, Y.-C., Yan, C.H., et al. (2017) Abnormal Subchondral Bone Remodeling and Its Association with Articular Cartilage Degradation in Knees of Type 2 Diabetes Patients. Bone Research, 5, 1-12.
https://doi.org/10.1038/boneres.2017.34
|
[76]
|
Franke, S., Rüster, C., Pester, J., et al. (2011) Advanced Glycation End Products Affect Growth and Function of Osteoblasts. Clinical and Experimental Rheumatology-Incl Supplements, 29, 650.
|
[77]
|
Hui, A.Y., Mccarty, W.J., Masuda, K., et al. (2012) A Systems Biology Approach to Synovial Joint Lu-brication in Health, Injury, and Disease. Wiley Interdisciplinary Reviews: Systems Biology and Medicine, 4, 15-37.
https://doi.org/10.1002/wsbm.157
|
[78]
|
Rhee, D.K., Marcelino, J., Baker, M., et al. (2005) The Secreted Glycopro-tein Lubricin Protects Cartilage Surfaces and Inhibits Synovial Cell Overgrowth. The Journal of Clinical Investigation, 115, 622-631.
https://doi.org/10.1172/JCI200522263
|
[79]
|
Dahl, L., Dahl, I., Engström-Laurent, A., et al. (1985) Concentration and Molecular Weight of Sodium Hyaluronate in Synovial Fluid from Patients with Rheumatoid Arthritis and Other Ar-thropathies. Annals of the Rheumatic Diseases, 44, 817-822. https://doi.org/10.1136/ard.44.12.817
|
[80]
|
Scanzello, C.R. and Goldring, S.R. (2012) The Role of Synovitis in Osteoarthritis Pathogenesis. Bone, 51, 249-257.
https://doi.org/10.1016/j.bone.2012.02.012
|
[81]
|
Tsai, C.-H., Chiang, Y.-C., Chen, H.-T., et al. (2013) High Glu-cose Induces Vascular Endothelial Growth Factor Production in Human Synovial Fibroblasts through Reactive Oxygen Species Generation. Biochimica et Biophysica Acta (BBA)-General Subjects, 1830, 2649-2658. https://doi.org/10.1016/j.bbagen.2012.12.017
|
[82]
|
Pessler, F., Dai, L., Diaz-Torne, C., et al. (2008) The Synovitis of “Non-Inflammatory” Orthopaedic Arthropathies: A Quantitative Histological and Immunohistochemical Analysis. Annals of the Rheumatic Diseases, 67, 1184-1187.
https://doi.org/10.1136/ard.2008.087775
|
[83]
|
Li, Q., Wen, Y., Wang, L., et al. (2021) Hyperglycemia-Induced Accumulation of Advanced Glycosylation End Products in Fibroblast-Like Synoviocytes Promotes Knee Osteoarthritis. Experimental & Molecular Medicine, 53, 1735- 1747. https://doi.org/10.1038/s12276-021-00697-6
|
[84]
|
Hamada, D., Maynard, R., Schott, E., et al. (2016) Insulin Suppresses TNF-Dependent Early Osteoarthritic Changes Associated with Obesity and Type 2 Diabetes. Arthritis & Rheumatology (Hoboken, NJ), 68, 1392.
https://doi.org/10.1002/art.39561
|
[85]
|
叶文春, 方向明, 王玉容, 等. 鼠神经生长因子联合硫辛酸治疗糖尿病周围神经病变的疗效观察[J]. 四川医学, 2013, 33(12): 2098-2100.
|
[86]
|
Zochodne, D.W. (2007) Diabetes Mellitus and the Peripheral Nervous System: Manifestations and Mechanisms. Muscle & Nerve: Official Journal of the American Association of Electrodiagnostic Medicine, 36, 144-166.
https://doi.org/10.1002/mus.20785
|
[87]
|
Leaverton, P.E., Peregoy, J., Fahlman, L., et al. (2012) Does Diabetes Hide Osteoarthritis Pain? Medical Hypotheses, 78, 471-474. https://doi.org/10.1016/j.mehy.2012.01.008
|
[88]
|
段俊虎. 2型糖尿病周围神经损害与膝关节骨关节炎严重程度的关系研究[J]. 中国医药导刊, 2015, 17(5): 462-464.
|
[89]
|
朱延波. 2型糖尿病周围神经损害对膝关节骨关节炎严重程度的影响[J]. 实用妇科内分泌电子杂志, 2016, 3(5): 133.
|