急性心肌梗死合并低T3综合征发生机制的研究进展
Research Progress on Pathogenesis and Harm of Acute Myocardial Infarction Complicated with Low T3 Syndrome
DOI: 10.12677/ACM.2021.113127, PDF,   
作者: 薛万腾, 吕志慧, 王营忠*:延安大学附属医院,陕西 延安
关键词: 急性心肌梗死低T3综合征Acute Myocardial Infarction Low T3 Syndrome
摘要: 急性心肌梗死目前是缺血性心脏病的主要病因及死亡原因,急性心肌梗死将导致甲状腺功能的急性改变,甲状腺激素在心脏血管生理病理过程中起着重要作用,急性心肌梗死时出现低T3综合征,低T3综合征进一步加重心肌损害,本文就其发生机制及危害性的研究进展做一综述。
Abstract: Acute myocardial infarction is the leading cause of ischemic heart disease and cause of death. Acute myocardial infarction will lead to acute alterations in thyroid function. Thyroid hormone in cardiac blood vessels plays an important role in physiological and pathological process. Low T3 syndrome occurs in acute myocardial infarction, and further aggravates myocardial damage. This paper summarizes the research progress of its occurrence mechanism and harm.
文章引用:薛万腾, 吕志慧, 王营忠. 急性心肌梗死合并低T3综合征发生机制的研究进展[J]. 临床医学进展, 2021, 11(3): 889-894. https://doi.org/10.12677/ACM.2021.113127

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