|
[1]
|
Saeedi, P., Petersohn, I., Salpea, P., et al. (2019) Global and Regional Diabetes Prevalence Estimates for 2019 and Pro-jections for 2030 and 2045: Results from the International Diabetes Federation Diabetes Atlas, 9th Edition. Diabetes Re-search and Clinical Practice, 157, Article ID: 107843. [Google Scholar] [CrossRef] [PubMed]
|
|
[2]
|
Yang, Z., Feng, L., Huang, Y. and Xia, N. (2019) A Differential Diagnosis Model for Diabetic Nephropathy and Non-Diabetic Renal Disease in Patients with Type 2 Diabetes Complicated with Chronic Kidney Disease. Diabetes, Metabolic Syn-drome and Obesity: Targets and Therapy, 12, 1963-1972. [Google Scholar] [CrossRef]
|
|
[3]
|
Hutton, H.L., Ooi, J.D., Holdsworth, S.R., et al. (2016) The NLRP3 Inflammasome in Kidney Disease and Autoimmunity. Nephrology, 21, 736-744. [Google Scholar] [CrossRef] [PubMed]
|
|
[4]
|
Cookson, B.T. and Brennan, M.A. (2001) Pro-Inflammatory Programmed Cell Death. Trends in Microbiology, 9, 113-114. [Google Scholar] [CrossRef]
|
|
[5]
|
Wang, K., Sun, Q., Zhong, X., et al. (2020) Structural Mechanism for GSDMD Targeting by Autoprocessed Caspases in Pyroptosis. Cell, 180, 941-955.E20. [Google Scholar] [CrossRef] [PubMed]
|
|
[6]
|
Komada, T. and Muruve, D.A. (2019) The Role of Inflammasomes in Kidney Disease. Nature Reviews Nephrology, 15, 501-520. [Google Scholar] [CrossRef] [PubMed]
|
|
[7]
|
Wu, M., Han, W., Song, S., et al. (2018) NLRP3 Deficiency Ameliorates Renal Inflammation and Fibrosis in Diabetic Mice. Molecular and Cellular Endocrinology, 478, 115-125. [Google Scholar] [CrossRef] [PubMed]
|
|
[8]
|
Gu, C.M., Liu, S.M., Wang, H.Y. and Dou, H.C. (2019) Role of the Thioredoxin Interacting Protein in Diabetic Nephropathy and the Mechanism of Regulating NOD-Like Receptor Protein 3 Inflammatory Corpuscle. International Journal of Molecular Medicine, 43, 2440-2450.
|
|
[9]
|
Li, W., Wu, Z., Ma, Q., et al. (2014) Hyperglycemia Regulates TXNIP/TRX/ROS Axis via p38 MAPK and ERK Pathways in Pancreatic Cancer. Current Cancer Drug Targets, 14, 348-356. [Google Scholar] [CrossRef] [PubMed]
|
|
[10]
|
An, X., Zhang, Y., Cao, Y., et al. (2020) Punicalagin Protects Diabetic Nephropathy by Inhibiting Pyroptosis Based on TXNIP/NLRP3 Pathway. Nutrients, 12, Article No. 1516. [Google Scholar] [CrossRef] [PubMed]
|
|
[11]
|
Gao, Y., Ma, Y., Xie, D. and Jiang, H. (2022) ManNAc Protects against Podocyte Pyroptosis via Inhibiting Mitochondrial Damage and ROS/NLRP3 Signaling Pathway in Diabetic Kid-ney Injury Model. International Immunopharmacology, 107, Article ID: 108711. [Google Scholar] [CrossRef] [PubMed]
|
|
[12]
|
Zuo, Y., Chen, L., Gu, H., He, X., Ye, Z., Wang, Z., Shao, Q. and Xue, C. (2021) GSDMD-Mediated Pyroptosis: A Critical Mechanism of Diabetic Nephropathy. Expert Reviews in Molecular Medicine, 23, E23. [Google Scholar] [CrossRef] [PubMed]
|
|
[13]
|
Yang, F., Qin, Y., Lv, J., et al. (2018) Silencing Long Non-Coding RNA Kcnq1ot1 Alleviates Pyroptosis and Fibrosis in Diabetic Cardiomyopathy. Cell Death & Disease, 9, Article No. 1000. [Google Scholar] [CrossRef] [PubMed]
|
|
[14]
|
Zheng, J., Zhang, S., Chen, H., et al. (2020) Pro-tosappanin-A and Oleanolic Acid Protect Injured Podocytes from Apoptosis through Inhibition of AKT-mTOR Signaling. Cell Biology International, 44, 189-199. [Google Scholar] [CrossRef] [PubMed]
|
|
[15]
|
Zhang, C., Gong, Y., Li, N., et al. (2020) Long Non-Coding RNA Kcnq1ot1 Promotes sC5b-9-Induced Podocyte Pyroptosis by Inhibiting miR-486a-3p and Upregulating NLRP3. Ameri-can Journal of Physiology-Cell Physiology, 320, C355-C364. [Google Scholar] [CrossRef] [PubMed]
|
|
[16]
|
Zuo, Y., Chen, L., He, X., et al. (2021) Atorvastatin Regulates MALAT1/miR-200c/NRF2 Activity to Protect against Podo-cyte Pyroptosis Induced by High Glucose. Diabetes, Metabolic Syndrome and Obesity: Targets and Therapy, 14, 1631-1645. [Google Scholar] [CrossRef]
|
|
[17]
|
Wang, C., Pan, Y., Zhang, Q.Y., et al. (2012) Quercetin and Allopurinol Ameliorate Kidney Injury in STZ-Treated Rats with Regulation of Renal NLRP3 Inflammasome Activa-tion and Lipid Accumulation. PLOS ONE, 7, e38285. [Google Scholar] [CrossRef] [PubMed]
|
|
[18]
|
Birnbaum, Y., Bajaj, M., Yang, H.C. and Ye, Y. (2018) Com-bined SGLT2 and DPP4 Inhibition Reduces the Activation of the Nlrp3/ASC Inflammasome and Attenuates the Devel-opment of Diabetic Nephropathy in Mice with Type 2 Diabetes. Cardiovascular Drugs and Therapy, 32, 135-145. [Google Scholar] [CrossRef] [PubMed]
|
|
[19]
|
Zhu, W., Li, Y.Y., Zeng, H.X., Liu, X.Q., Sun, Y.T., Jiang, L., Xia, L.L. and Wu, Y.G. (2021) Carnosine Alleviates Podocyte Injury in Diabeticnephropathy by Targeting Caspa-se-1-Mediated Pyroptosis. International Immunopharmacology, 101, Article ID: 108236. [Google Scholar] [CrossRef] [PubMed]
|
|
[20]
|
Bergsbaken, T., Fink, S.L. and Cookson, B.T. (2009) Pyropto-sis: Host Cell Death and Inflammation. Nature Reviews Microbiology, 7, 99-109. [Google Scholar] [CrossRef] [PubMed]
|
|
[21]
|
Shi, J., Zhao, Y., Wang, Y., et al. (2014) Inflammatory Caspases Are Innate Immune Receptors for Intracellular LPS. Nature, 514, 187-192. [Google Scholar] [CrossRef] [PubMed]
|
|
[22]
|
Kayagaki, N., Stowe, I.B., Lee, B.L., O’Rourke, K., Anderson, K., Warming, S., Cuellar, T., Haley, B., Roose-Girma, M., Phung, Q.T., Liu, P.S., Lill, J.R., Li, H., Wu, J., Kummerfeld, S., Zhang, J., Lee, W.P., Snipas, S.J., Salvesen, G.S., Morris, L.X., Fitzgerald, L., Zhang, Y., Bertram, E.M., Goodnow, C.C. and Dixit, V.M. (2015) Caspase-11 Cleaves Gasdermin D for Non-Canonical Inflammasome Signalling. Nature, 526, 666-671. [Google Scholar] [CrossRef] [PubMed]
|
|
[23]
|
Yang, D., He, Y., MuOz-Planillo, R., et al. (2015) Caspase-11 Requires the Pannexin-1 Channel and the Purinergic P2X7 Pore to Mediate Pyroptosis and Endotoxic Shock. Immunity, 43, 923-932. [Google Scholar] [CrossRef] [PubMed]
|
|
[24]
|
Cheng, Q., Pan, J., Zhou, Z.L., et al. (2021) Caspase-11/4 and Gasdermin D-Mediated Pyroptosis Contributes to Podocyte Injury in Mouse Diabetic Nephropathy. Acta Pharmacolog-ica Sinica, 42, 954-963. [Google Scholar] [CrossRef] [PubMed]
|
|
[25]
|
Xie, C., Wu, W., Tang, A., Luo, N. and Tan, Y. (2019) lncRNA GAS5/miR-452-5p Reduces Oxidative Stress and Pyroptosis of High-Glucose-Stimulated Renal Tubular Cells. Diabetes, Metabolic Syndrome and Obesity: Targets and Therapy, 12, 2609-2617. [Google Scholar] [CrossRef]
|
|
[26]
|
Napier, B.A., Brubaker, S.W., Sweeney, T.E., et al. (2016) Com-plement Pathway Amplifies Caspase-11-Dependent Cell Death and Endotoxin-Induced Sepsis Severity. Journal of Ex-perimental Medicine, 213, 2365-2382. [Google Scholar] [CrossRef] [PubMed]
|
|
[27]
|
Ma, J., Chadban, S.J., Zhao, C.Y., et al. (2014) TLR4 Activation Pro-motes Podocyte Injury and Interstitial Fibrosis in Diabetic Nephropathy. PLOS ONE, 9, e97985. [Google Scholar] [CrossRef] [PubMed]
|
|
[28]
|
Wang, Y., Gao, W., Shi, X., et al. (2017) Chemotherapy Drugs Induce Pyroptosis through Caspase-3 Cleavage of a Gasdermin. Nature, 547, 99-103. [Google Scholar] [CrossRef] [PubMed]
|
|
[29]
|
Rogers, C., Fernandes-Alnemri, T., Mayes, L., et al. (2017) Cleavage of DFNA5 by Caspase-3 during Apoptosis Mediates Progression to Secondary Necrotic/Pyroptotic Cell Death. Nature Communications, 8, Article No. 14128. [Google Scholar] [CrossRef] [PubMed]
|
|
[30]
|
Xia, W., Li, Y., Wu, M., et al. (2021) Gasdermin E Deficiency Atten-uates Acute Kidney Injury by Inhibiting Pyroptosis and Inflammation. Cell Death & Disease, 12, Article No. 139. [Google Scholar] [CrossRef] [PubMed]
|
|
[31]
|
Wen, S., Wang, Z.H., Zhang, C.X., et al. (2020) Caspase-3 Promotes Diabetic Kidney Disease through Gasdermin E- Mediated Progression to Secondary Necrosis during Apopto-sis. Diabetes, Metabolic Syndrome and Obesity: Targets and Therapy, 13, 313-323. [Google Scholar] [CrossRef]
|
|
[32]
|
Xu, W.F., Zhang, Q., Ding, C.J., et al. (2021) Gasdermin E-Derived Caspase-3 Inhibitors Effectively Protect Mice from Acute Hepatic Failure. Acta Pharmacologica Sinica, 42, 68-76. [Google Scholar] [CrossRef] [PubMed]
|