浅谈Déjà vu现象的研究进展
Brief Introduction to the Research Progress of Déjà vu Phenomenon
DOI: 10.12677/ACM.2023.1381852, PDF,   
作者: 王爱元*, 尹 争:青海大学研究生院,青海 西宁;李 沛:青海省人民医院神经内科,青海 西宁
关键词: Déjà vu神经精神错误记忆脑电图基因治疗Déjà vu Neuropsychiatry False Memory Electroencephalogram Gene Treatment
摘要: Déjà vu是一种法语表达,即似曾相识的意思。多达97%的人口在一生中至少经历过一次Déjà vu,67%的人经常经历这种情况。Déjà vu现象考虑可能是一种残存的记忆,在某种程度上是大脑功能的“小故障”,类似于视觉错觉,其实是大脑工作中产生的错误记忆。为了理解Déjà vu是如何发生的,我们拟从神经解剖、临床表现特点、脑电图模式、基因分析以及相关治疗来更详细地探索Déjà vu。
Abstract: Déjà vu is a French expression, which means I’ve seen something before. As many as 97 percent of the population has experienced Déjà vu at least once in their lifetime, with 67 percent experiencing it regularly. The Déjà vu phenomenon is considered to be a vestigial memory that is in some way a “glitch” in brain function, akin to an optical illusion that is actually a false memory created by the brain at work. In order to understand how Déjà vu occurs, we intend to explore Déjà vu in more de-tail in terms of neuroanatomy, clinical features, EEG patterns, genetic analysis, and related treat-ments.
文章引用:王爱元, 李沛, 尹争. 浅谈Déjà vu现象的研究进展[J]. 临床医学进展, 2023, 13(8): 13254-13259. https://doi.org/10.12677/ACM.2023.1381852

参考文献

[1] O’Connor, A.R. and Moulin, C.J.A. (2010) Recognition without Identification, Erroneous Familiarity, and Déjà Vu. Current Psychiatry Reports, 12, 165-173. [Google Scholar] [CrossRef] [PubMed]
[2] Urquhart, J.A., Siva-kumaran, M.H., Macfarlane, J.A. and O’Connor, A.R. (2018) fMRI Evidence Supporting the Role of Memory Conflict in the Déjà Vu Experience. Memory, 29, 921-932. [Google Scholar] [CrossRef] [PubMed]
[3] O’Connor, A.R. and Moulin, C.J.A. (2008) The Persistence of Erroneous Familiarity in an Epileptic Male: Challenging Perceptual Theories of Déjà Vu Activation. Brain and Cogni-tion, 68, 144-147. [Google Scholar] [CrossRef] [PubMed]
[4] Wells, C.E., O’Connor, A.R. and Moulin, C.J.A. (2018) Déjà Vu Experiences in Anxiety. Memory, 29, 895-903. [Google Scholar] [CrossRef] [PubMed]
[5] Brázdil, M., Mareček, R., Urbánek, T., et al. (2012) Unveil-ing the Mystery of Déjà Vu: The Structural Anatomy of Déjà Vu. Cortex, 48, 1240-1243. [Google Scholar] [CrossRef] [PubMed]
[6] Labate, A., Cerasa, A., Mumoli, L., et al. (2014) Neu-ro-Anatomical Differences among Epileptic and Non-Epileptic Déjà-Vu. Cortex, 64, 1-7. [Google Scholar] [CrossRef] [PubMed]
[7] Shaw, D.J., Mareček, R. and Brázdil, M. (2016) Structural Co-variance Mapping Delineates Medial and Medio-Lateral Temporal Networks in Déjà Vu. Brain Imaging and Behavior, 10, 1068-1079. [Google Scholar] [CrossRef] [PubMed]
[8] Qiu, J., Xia, Y., He, L., et al. (2018) Abnormal rsFC and GMV Changes in Parahippocampal and DLPFC for High Déjà Vu Experienced Subjects. Biological Psychology, 133, 72-78. [Google Scholar] [CrossRef] [PubMed]
[9] Li, W., Li, Y., Yang, W., et al. (2015) Brain Structures and Functional Connectivity Associated with Individual Differences in Internet Tendency in Healthy Young Adults. Neuro-psychologia, 70, 134-144. [Google Scholar] [CrossRef] [PubMed]
[10] Henson, R.N.A., Shallice, T. and Dolan, R.J. (1999) Right Prefrontal Cortex and Episodic Memory Retrieval: A Functional MRI Test of the Monitoring Hypothesis. Brain, 122, 1367-1381. [Google Scholar] [CrossRef] [PubMed]
[11] Fletcher, P.C. and Henson, R.N.A. (2001) Frontal Lobes and Human Memory: Insights from Functional Neuroimaging. Brain, 124, 849-881. [Google Scholar] [CrossRef] [PubMed]
[12] O’Connor, A.R., Barnier, A.J. and Cox, R.E. (2008) Déjà Vu in the Laboratory: A Behavioral and Experiential Comparison of Posthypnotic Amnesia and Posthypnotic Familiarity. The In-ternational Journal of Clinical and Experimental Hypnosis, 56, 425-450. [Google Scholar] [CrossRef] [PubMed]
[13] Sang, F.Y.P., Jáuregui-Renaud, K., Green, D.A., Bronstein, A.M. and Gresty, M.A. (2006) Depersonalisation/Derealisation Symptoms in Vestibular Disease. Journal of Neurology, Neurosurgery, and Psychiatry, 77, 760-766. [Google Scholar] [CrossRef] [PubMed]
[14] Brown, A.S. (2003) A Review of the Déjà Vu Experience. Psycho-logical Bulletin, 129, 394-413. [Google Scholar] [CrossRef] [PubMed]
[15] Cleary, A.M., Brown, A.S., Sawyer, B.D., et al. (2012) Famili-arity from the Configuration of Objects in 3-Dimensional Space and Its Relation to Déjà Vu: A Virtual Reality Investiga-tion. Consciousness and Cognition, 21, 969-975. [Google Scholar] [CrossRef] [PubMed]
[16] Adachi, N., Akanuma, N., Ito, M., et al. (2010) Two Forms of Déjà Vu Experiences in Patients with Epilepsy. Epilepsy & Behavior, 18, 218-222. [Google Scholar] [CrossRef] [PubMed]
[17] Palmini, A. and Gloor, P. (1992) The Localizing Value of Auras in Partial Seizures: A Prospective and Retrospective Study. Neurology, 42, 801-808. [Google Scholar] [CrossRef
[18] Berkovic, S.F., McIntosh, A., Howell, R.A., Mitchell, A., Sheffield, L.J. and Hopper, J.L. (1996) Familial Temporal Lobe Epilepsy: A Common Disorder Identified in Twins. Annals of Neurology, 40, 227-235. [Google Scholar] [CrossRef] [PubMed]
[19] Striano, P., Gambardella, A., Coppola, A., et al. (2008) Familial Mesi-al Temporal Lobe Epilepsy (FMTLE): A Clinical and Genetic Study of 15 Italian Families. Journal of Neurology, 255, 16-23. [Google Scholar] [CrossRef] [PubMed]
[20] Halgren, E., Walter, R.D., Cherlow, D.G. and Crandall, P.H. (1978) Mental Phenomena Evoked by Electrical Stimulation of the Human Hippocampal Formation and Amygdala. Brain, 101, 83-115. [Google Scholar] [CrossRef] [PubMed]
[21] Stevens, J.R. (1990) Psychiatric Consequences of Tem-poral Lobectomy for Intractable Seizures: A 20 - 30-Year Follow-Up of 14 Cases. Psychological Medicine, 20, 529-545. [Google Scholar] [CrossRef
[22] Penfield, W. (1955) The Twenty-Ninth Maudsley Lecture: The Role of the Temporal Cortex in Certain Psychical Phenomena. The Journal of Mental Science, 101, 451-465. [Google Scholar] [CrossRef] [PubMed]
[23] Petrusic, I., Pavlovski, V., Vucinic, D. and Jancic, J. (2014) Features of Migraine Aura in Teenagers. The Journal of Headache and Pain, 15, Article No. 87. [Google Scholar] [CrossRef] [PubMed]
[24] Vlasov, P.-N., Cherviakov, A.V. and Gnezdinsiĭ, V.V. (2013) [Electroencephalographic Characteristics of the Déjà Vu Phenomenon]. Zhurnal Nevrologii i Psikhiatrii Imeni S.S. Kor-sakova, 113, 3-9. (In Russian)
[25] Spatt, J. (2002) Déjà Vu: Possible Parahippocampal Mechanisms. The Journal of Neuropsychiatry and Clinical Neurosciences, 14, 6-10. [Google Scholar] [CrossRef] [PubMed]
[26] Achim, A.M. and Lepage, M. (2005) Neural Correlates of Memory for Items and for Associations: An Event-Related Functional Magnetic Resonance Imaging Study. Journal of Cognitive Neuroscience, 17, 652-667. [Google Scholar] [CrossRef] [PubMed]
[27] Pašić, M.B., Velić, E.H., Fotak, L., et al. (2018) Many Faces of Déjà Vu: A Narrative Review. Psychiatria Danubina, 30, 21-25.
[28] Bartolomei, F., Barbeau, E., Gavaret, M., et al. (2004) Cortical Stimulation Study of the Role of Rhinal Cortex in Déjà Vu and Reminiscence of Memories. Neurology, 63, 858-864. [Google Scholar] [CrossRef
[29] Corr, P.J. and Mobbs, D. (2018) From Epiphenomenon to Biologically Important Phenomena. Personality Neuroscience, 1, e1. [Google Scholar] [CrossRef] [PubMed]
[30] Gloor, P. (1990) Experiential Phenomena of Temporal Lobe Epilepsy: Facts and Hypotheses. Brain, 113, 1673-1694. [Google Scholar] [CrossRef] [PubMed]
[31] Bancaud, J., Brunet-Bourgin, F., Chauvel, P. and Halgren, E. (1994) Anatomical Origin of Déjà Vu and Vivid ‘Memories’ in Human Temporal Lobe Epilepsy. Brain, 117, 71-90. [Google Scholar] [CrossRef] [PubMed]
[32] Fertig, E., Lincoln, A., Martinuzzi, A., Mattson, R.H. and Hisama, F.M. (2003) Novel LGI1 Mutation in a Family with Autosomal Dominant Partial Epilepsy with Auditory Features. Neurology, 60, 1687-1690. [Google Scholar] [CrossRef
[33] Kalachikov, S., Evgrafov, O., Ross, B., et al. (2002) Mutations in LGI1 Cause Autosomal-Dominant Partial Epilepsy with Auditory Features. Nature Genetics, 30, 335-341. [Google Scholar] [CrossRef] [PubMed]
[34] Morante-Redolat, J.M., Gorostidi-Pagola, A., Piquer-Sirerol, S., et al. (2002) Mutations in the LGI1/Epitempin Gene on 10q24 Cause Autosomal Dominant Lateral Temporal Epilepsy. Human Mo-lecular Genetics, 11, 1119-1128. [Google Scholar] [CrossRef] [PubMed]
[35] Michelucci, R., Poza, J.J., Sofia, V., et al. (2003) Autosomal Dominant Lateral Temporal Epilepsy: Clinical Spectrum, New Epitempin Mutations, and Genetic Heterogeneity in Seven European Families. Epilepsia, 44, 1289-1297. [Google Scholar] [CrossRef] [PubMed]
[36] Compan, V., Daszuta, A., Salin, P., Sebben, M., Bockaert, J. and Dumuis, A. (1996) Lesion Study of the Distribution of Serotonin 5-HT4 Receptors in Rat Basal Ganglia and Hip-pocampus. The European Journal of Neuroscience, 8, 2591-2598. [Google Scholar] [CrossRef] [PubMed]
[37] Cai, X., Flores-Hernandez, J., Feng, J. and Yan, Z. (2002) Activity-Dependent Bidirectional Regulation of GABAA Receptor Channels by the 5-HT4 Receptor-Mediated Signalling in Rat Prefrontal Cortical Pyramidal Neurons. The Journal of Physiology, 540, 743-759. [Google Scholar] [CrossRef] [PubMed]
[38] Vilaró, M.T., Cortés, R. and Mengod, G. (2005) Serotonin 5-HT4 Receptors and Their mRNAs in Rat and Guinea Pig Brain: Distribution and Effects of Neurotoxic Lesions. Jour-nal of Comparative Neurology, 484, 418-439. [Google Scholar] [CrossRef] [PubMed]
[39] Nørgaard, M., Beliveau, V., Ganz, M., et al. (2021) A High-Resolution in Vivo Atlas of the Human Brain’s Benzodiazepine Binding Site of GABAA Receptors. Neuroimage, 232, Article ID: 117878. [Google Scholar] [CrossRef] [PubMed]